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transam nf κb p65 chemi tran scription factor assay kit  (Active Motif)

 
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    Active Motif transam nf κb p65 chemi tran scription factor assay kit
    Transam Nf κb P65 Chemi Tran Scription Factor Assay Kit, supplied by Active Motif, used in various techniques. Bioz Stars score: 96/100, based on 1587 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Average 96 stars, based on 1587 article reviews
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    transam nf κb p65 chemi tran scription factor assay kit  (Active Motif)
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    LMO2 may regulate NFKB pathway via Bex1 in ccRCC. A. Heatmap of differentially expressed genes in RNA sequencing data with LMO2 overexpression; B. GO, BP and MF analysis based on data in A; C. The enrichment of KEGG pathway implies that TNF-NFKB pathway is responsible for LMO2 regulation; D. The <t>p-p65</t> expression in LMO2 expression and control in 786-O and ACHN; E. Statistical analysis of OD value in D; F. The ELISA assay results of nuclear p-p65 concentration in LMO2 expression and control in 786-O and ACHN; G. String database indicates the possible interacting genes of LMO2. Organisms column was restricted to Homo sapiens; H. Gene Mania indicates the possible interacting genes; I. The BEX1 expression in ccRCC from TCGA database; J. The correlation analysis of BEX1 and LMO2 in ccRCC from the database TCGA. * P < 0.05, ⁎⁎ P < 0.01, ⁎⁎⁎ P < 0.001.
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    transam nf κb p65 kit  (Active Motif)
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    LMO2 may regulate NFKB pathway via Bex1 in ccRCC. A. Heatmap of differentially expressed genes in RNA sequencing data with LMO2 overexpression; B. GO, BP and MF analysis based on data in A; C. The enrichment of KEGG pathway implies that TNF-NFKB pathway is responsible for LMO2 regulation; D. The <t>p-p65</t> expression in LMO2 expression and control in 786-O and ACHN; E. Statistical analysis of OD value in D; F. The ELISA assay results of nuclear p-p65 concentration in LMO2 expression and control in 786-O and ACHN; G. String database indicates the possible interacting genes of LMO2. Organisms column was restricted to Homo sapiens; H. Gene Mania indicates the possible interacting genes; I. The BEX1 expression in ccRCC from TCGA database; J. The correlation analysis of BEX1 and LMO2 in ccRCC from the database TCGA. * P < 0.05, ⁎⁎ P < 0.01, ⁎⁎⁎ P < 0.001.
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    LMO2 may regulate NFKB pathway via Bex1 in ccRCC. A. Heatmap of differentially expressed genes in RNA sequencing data with LMO2 overexpression; B. GO, BP and MF analysis based on data in A; C. The enrichment of KEGG pathway implies that TNF-NFKB pathway is responsible for LMO2 regulation; D. The <t>p-p65</t> expression in LMO2 expression and control in 786-O and ACHN; E. Statistical analysis of OD value in D; F. The ELISA assay results of nuclear p-p65 concentration in LMO2 expression and control in 786-O and ACHN; G. String database indicates the possible interacting genes of LMO2. Organisms column was restricted to Homo sapiens; H. Gene Mania indicates the possible interacting genes; I. The BEX1 expression in ccRCC from TCGA database; J. The correlation analysis of BEX1 and LMO2 in ccRCC from the database TCGA. * P < 0.05, ⁎⁎ P < 0.01, ⁎⁎⁎ P < 0.001.
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    LMO2 may regulate NFKB pathway via Bex1 in ccRCC. A. Heatmap of differentially expressed genes in RNA sequencing data with LMO2 overexpression; B. GO, BP and MF analysis based on data in A; C. The enrichment of KEGG pathway implies that TNF-NFKB pathway is responsible for LMO2 regulation; D. The <t>p-p65</t> expression in LMO2 expression and control in 786-O and ACHN; E. Statistical analysis of OD value in D; F. The ELISA assay results of nuclear p-p65 concentration in LMO2 expression and control in 786-O and ACHN; G. String database indicates the possible interacting genes of LMO2. Organisms column was restricted to Homo sapiens; H. Gene Mania indicates the possible interacting genes; I. The BEX1 expression in ccRCC from TCGA database; J. The correlation analysis of BEX1 and LMO2 in ccRCC from the database TCGA. * P < 0.05, ⁎⁎ P < 0.01, ⁎⁎⁎ P < 0.001.
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    LMO2 may regulate NFKB pathway via Bex1 in ccRCC. A. Heatmap of differentially expressed genes in RNA sequencing data with LMO2 overexpression; B. GO, BP and MF analysis based on data in A; C. The enrichment of KEGG pathway implies that TNF-NFKB pathway is responsible for LMO2 regulation; D. The <t>p-p65</t> expression in LMO2 expression and control in 786-O and ACHN; E. Statistical analysis of OD value in D; F. The ELISA assay results of nuclear p-p65 concentration in LMO2 expression and control in 786-O and ACHN; G. String database indicates the possible interacting genes of LMO2. Organisms column was restricted to Homo sapiens; H. Gene Mania indicates the possible interacting genes; I. The BEX1 expression in ccRCC from TCGA database; J. The correlation analysis of BEX1 and LMO2 in ccRCC from the database TCGA. * P < 0.05, ⁎⁎ P < 0.01, ⁎⁎⁎ P < 0.001.
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    transam nf-κb p65 elisa kit  (Active Motif)
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    LMO2 may regulate NFKB pathway via Bex1 in ccRCC. A. Heatmap of differentially expressed genes in RNA sequencing data with LMO2 overexpression; B. GO, BP and MF analysis based on data in A; C. The enrichment of KEGG pathway implies that TNF-NFKB pathway is responsible for LMO2 regulation; D. The <t>p-p65</t> expression in LMO2 expression and control in 786-O and ACHN; E. Statistical analysis of OD value in D; F. The ELISA assay results of nuclear p-p65 concentration in LMO2 expression and control in 786-O and ACHN; G. String database indicates the possible interacting genes of LMO2. Organisms column was restricted to Homo sapiens; H. Gene Mania indicates the possible interacting genes; I. The BEX1 expression in ccRCC from TCGA database; J. The correlation analysis of BEX1 and LMO2 in ccRCC from the database TCGA. * P < 0.05, ⁎⁎ P < 0.01, ⁎⁎⁎ P < 0.001.
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    LMO2 may regulate NFKB pathway via Bex1 in ccRCC. A. Heatmap of differentially expressed genes in RNA sequencing data with LMO2 overexpression; B. GO, BP and MF analysis based on data in A; C. The enrichment of KEGG pathway implies that TNF-NFKB pathway is responsible for LMO2 regulation; D. The <t>p-p65</t> expression in LMO2 expression and control in 786-O and ACHN; E. Statistical analysis of OD value in D; F. The ELISA assay results of nuclear p-p65 concentration in LMO2 expression and control in 786-O and ACHN; G. String database indicates the possible interacting genes of LMO2. Organisms column was restricted to Homo sapiens; H. Gene Mania indicates the possible interacting genes; I. The BEX1 expression in ccRCC from TCGA database; J. The correlation analysis of BEX1 and LMO2 in ccRCC from the database TCGA. * P < 0.05, ⁎⁎ P < 0.01, ⁎⁎⁎ P < 0.001.
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    LMO2 may regulate NFKB pathway via Bex1 in ccRCC. A. Heatmap of differentially expressed genes in RNA sequencing data with LMO2 overexpression; B. GO, BP and MF analysis based on data in A; C. The enrichment of KEGG pathway implies that TNF-NFKB pathway is responsible for LMO2 regulation; D. The <t>p-p65</t> expression in LMO2 expression and control in 786-O and ACHN; E. Statistical analysis of OD value in D; F. The ELISA assay results of nuclear p-p65 concentration in LMO2 expression and control in 786-O and ACHN; G. String database indicates the possible interacting genes of LMO2. Organisms column was restricted to Homo sapiens; H. Gene Mania indicates the possible interacting genes; I. The BEX1 expression in ccRCC from TCGA database; J. The correlation analysis of BEX1 and LMO2 in ccRCC from the database TCGA. * P < 0.05, ⁎⁎ P < 0.01, ⁎⁎⁎ P < 0.001.
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    LMO2 may regulate NFKB pathway via Bex1 in ccRCC. A. Heatmap of differentially expressed genes in RNA sequencing data with LMO2 overexpression; B. GO, BP and MF analysis based on data in A; C. The enrichment of KEGG pathway implies that TNF-NFKB pathway is responsible for LMO2 regulation; D. The p-p65 expression in LMO2 expression and control in 786-O and ACHN; E. Statistical analysis of OD value in D; F. The ELISA assay results of nuclear p-p65 concentration in LMO2 expression and control in 786-O and ACHN; G. String database indicates the possible interacting genes of LMO2. Organisms column was restricted to Homo sapiens; H. Gene Mania indicates the possible interacting genes; I. The BEX1 expression in ccRCC from TCGA database; J. The correlation analysis of BEX1 and LMO2 in ccRCC from the database TCGA. * P < 0.05, ⁎⁎ P < 0.01, ⁎⁎⁎ P < 0.001.

    Journal: Translational Oncology

    Article Title: LMO2 confers value as a potential immunotherapy marker in pan-cancer analysis and inhibits progression of Clear Cell Renal Cell Carcinoma

    doi: 10.1016/j.tranon.2025.102409

    Figure Lengend Snippet: LMO2 may regulate NFKB pathway via Bex1 in ccRCC. A. Heatmap of differentially expressed genes in RNA sequencing data with LMO2 overexpression; B. GO, BP and MF analysis based on data in A; C. The enrichment of KEGG pathway implies that TNF-NFKB pathway is responsible for LMO2 regulation; D. The p-p65 expression in LMO2 expression and control in 786-O and ACHN; E. Statistical analysis of OD value in D; F. The ELISA assay results of nuclear p-p65 concentration in LMO2 expression and control in 786-O and ACHN; G. String database indicates the possible interacting genes of LMO2. Organisms column was restricted to Homo sapiens; H. Gene Mania indicates the possible interacting genes; I. The BEX1 expression in ccRCC from TCGA database; J. The correlation analysis of BEX1 and LMO2 in ccRCC from the database TCGA. * P < 0.05, ⁎⁎ P < 0.01, ⁎⁎⁎ P < 0.001.

    Article Snippet: NF-κB p65 activation was measured by a TransAM NF-κB p65 kit (Active Motif, USA).

    Techniques: RNA Sequencing, Over Expression, Expressing, Control, Enzyme-linked Immunosorbent Assay, Concentration Assay